Brady Weissbourd named Klingenstein-Simons Fellow
Three-year fellowship will support Weissbourd’s research on how the C. hemisphaerica jellyfish survives and thrives by constantly making new neurons.
Three-year fellowship will support Weissbourd’s research on how the C. hemisphaerica jellyfish survives and thrives by constantly making new neurons.
Distinctive EEG patterns indicate when a patient’s state of unconsciousness under general anesthesia is more profound than necessary.
Prestigious awards recognize community support of MIT’s goals, values, and mission.
Scientists find a protein common to flies and people is essential for supporting the structure of axons that neurons project to make circuit connections.
MIT researchers characterize gene expression patterns for 22,500 brain vascular cells across 428 donors, revealing insights for Alzheimer’s onset and potential treatments.
Symposium speakers describe numerous ways to promote prevention, resilience, healing, and wellness after early-life stresses.
Tactile stimulation improved motor performance, reduced phosphorylated tau, preserved neurons and synapses, and reduced DNA damage, a new study shows.
A full-building energy efficiency project aims to reduce total campus emissions by 2 percent.
With full genetic control and visibility into neural activity and behavior, MIT scientists map out chemical’s role in behavior.
Developing a new neuroscience model is no small feat. New faculty member Brady Weissbourd has risen to the challenge in order to study nervous system evolution, development, regeneration, and function.
New MIT faculty member investigates how sensory input from within the body controls mammalian physiology and behavior.
Seven staff members honored for their dedication to the School of Science and to the Institute.
A new study tests an alternative to external stimulation for measuring when subjects lose and regain responsiveness during sedation and anesthesia.
Neurons that form part of a memory circuit are among the first brain cells to show signs of neurodegeneration in Alzheimer’s disease.
The peptide blocks a hyperactive brain enzyme that contributes to the neurodegeneration seen in Alzheimer’s and other diseases.