MIT affiliates named 2026 Schmidt Sciences AI2050 Fellows
Five MIT faculty members and two additional alumni are honored with fellowships to advance research on beneficial AI.
Five MIT faculty members and two additional alumni are honored with fellowships to advance research on beneficial AI.
By tracking over 1 million cells, researchers created a detailed map of embryonic development in mice. It could provide development and disease insights.
Professor Michael Yaffe and six MIT alumni are honored for their outstanding professional achievement and commitment to service.
A new computational approach reveals how cells communicate to assemble into tissues. This could accelerate stem cell engineering, improve disease models, and inform therapies.
Some tumor cells develop resistance by amplifying the KRAS gene, while others change their tumor type, MIT researchers have found.
They also found that obesity may put patients at higher risk for activation of the pathway. Drugs that block the pathway may help prevent metastasis.
This summer, BSG-MSRP-Bio student Marina Milea investigated how lung cancers become resistant to targeted therapies, gaining hands-on research experience in the Jacks Lab at the Koch Institute.
The Institute welcomes its first cohort of QMIT Fellows this fall to advance interdisciplinary quantum research.
A new machine-learning framework aims to improve the success rate of computational protein design while moving away from results that reproduce sequences found in nature.
This discovery points toward new combination strategies against cancer, and may explain the iron buildup seen in disorders such as early-onset Parkinson’s disease.
A new study by Whitehead Institute researchers identifies how the widespread parasite Toxoplasma gondii survives in crowded environments in infected cells, which helps it persist in long-lasting brain cysts.
A study of worms finds a protein that helps shape the structure of the genome is critical for establishing the identity of some neurons.
Tonegawa made landmark discoveries about how the immune system generates antibody diversity and how the brain forms memories.
MIT researchers have found that a ketogenic high-fat, low-carbohydrate diet fuels the growth of intestinal tumors.
In certain species of bacteria, the answer lies in shielding RNA transcripts from a quality-control factor called Rho. Understanding the requirements for expressible sequences is critical for expression engineering of therapeutic agents.